Special Issue: Advances in Molecular Neuropathology

This special issue belongs to the section Molecular Pathology
International Journal of Molecular Sciences (ISSN 1422-0067)

[Editors] [Announced Papers] [Published Papers] [List of Keywords] [Manuscript Submission]


 Manuscript Submisison Deadline: 15 December 2008

 

Editors:

Editorial Office
IJMS Editorial Office
Molecular Diversity Preservation International
Kandererstrasse 25, CH-4057 Basel, Switzerland
Tel. +41-61-683-7734; Fax: +41-61-302-8918
Address manuscript submissions to: Dr. Yuan Gao, E-mail: [email protected]

Guest Editor
Prof. Dr. Kurt A. Jellinger
Institute of Clinical Neurobiology, Kenyongasse 18, A-1070 Vienna, Austria
Tel. +43-1-5266534; Fax: +43-1-5266534
E-mail: [email protected]

Rationale for the special issue on "Advances in Molecular Neuropathology"

Dear Colleagues,

During the recent years, considerable progress has been achieved in molecular genetics, pathogenesis and pathology of many nervous system disorders, based on new and modern technologies and animal models.

In the field of tumors of the CSN, the new 2007 classification, based on modern molecular genetics, immunocytochemistry and proteomics, has provided deeper insights into the pathogenesis and molecular pathobiology of CNS malignancies and has listed several new entities to the existing grading system, thus broadening the diagnostic spectrum and prognostic factors.

Modern research in inflammatory disorders of the nervous system has been focused on different types of multiple sclerosis (MS), their molecular and immunopathology, as well as on new molecular genetic findings and animal models of MS and related disorders, such as neuromyelitis optica and immun-mediated neuropathies, neuro-AIDS (CNS-HIV), and the relations between neuroinflammation and neurodegeneration.

One of the fields of most intensive and successful research is neurodegeneration and dementias, where many new phenotypes have been detected and classified using modern molecular genetics and animal and cellular models. Although the etiology and pathogenesis of Alzheimer disease and many other neurodegenerative and dementing disorders are still far from being elucidated, recent studies have provided important and meaningful results about the role of protein misfolding in neurodegeneration, eg, in the relationship of β-amyloid and tau protein in the pathogenesis of brain aging and Alzheimer disease, the role of α-synuclein and tau in neurodegeneration. Modern research has detected new forms of neurodegenerative disorders, such as the TDP-43-pathies, but has also provided new insights into the pathogenesis and molecular pathology of well-known entities, such as CAG repeat disorders, motor neuron diseases, prion diseases, peroxisomal and mitochondrial disorders.

The present special issue will present various aspects of modern research in molecular neuropathology in order to provide new impacts for further research in this up-to-date field of neurosciences.

Kurt A. Jellinger

Keywords:
  • molecular neuropathology
  • molecular genetics
  • pathogenesis of nervous system disorders
  • classification of CNS tumors
  • neuroinflammation
  • multiple sclerosis
  • neuro-AIDS
  • neurodegenerative diseases
  • dementias
  • Alzheimer disease
  • protein misfolding diseases
  • prion diseases
  • synucleinopathies
  • tauopathies
Call for Papers: download the call for paper message here

Manuscript Submission:

You are invited to submit your manuscript by e-mail to [email protected] with copy to the guest editor. Papers will be published continuously until the deadline and listed together at this special issue website.

Manuscript Submisison Deadline: 15 December 2008

The International Journal of Molecular Sciences (http://www.mdpi.org/ijms/; ISSN 1422-0067) was launched in 2000 and is an Open Access journal. According to SCI database, the IJMS has an impact factor (cites per article) of 0.750 in 2007. The IJMS maintains a rapid editorial procedure and a rigorous peer-review system. Well written papers have been peer-reviewed and published in less than 4 weeks from manuscript submission. This journal is abstracted and indexed very rapidly by Chemical Abstracts, the Science Citation Index Expanded, the Chemistry Citation Index, EMBASE, Scopus and Google Scholar. The IJMS has been accepted for coverage in PubMed/MEDLINE starting the 2008 volume. Articles from this volume will appear in PubMed by end of November 2008. Additionally, all articles will be archived in PubMed Central.

Submitted papers should not have been published previously, nor be currently under consideration for publication elsewhere. Please visit the Instructions for Authors at http://www.mdpi.org/ijms/publguid.htm before submitting a paper. Open Access publication fees are 800 CHF per paper. English correction fees (250 CHF) will be added in certain cases (1050 CHF per paper for those papers that require extensive additional formatting and/or English corrections).

Announced Papers:
 
Type of Paper: Article
Title: Hypercholesterolemia Effect on Brain-to-blood Clearance of Fluorescein-labeled ABeta
Authors: Thomas G. Beach
Affiliations: Sun Health Research Institute, 10515 West Santa Fe Drive, Sun City, AZ 85372; Tel. 623 876-5643; Fax: 623 815-2967; E-Mail: [email protected]
Abstact: Multiple risk factors for atherosclerotic vascular disease (AVD) have been shown to also be risk factors for Alzheimer's disease (AD). A molecular mechanism for this has not been convincingly identified. We have hypothesized that AVD is associated wtih brain microvascular inflammation that impairs the normal brain-to-blood clearance of Abeta. This hypothesis was tested in rabbits fed high cholesterol and control diets and injected into the lateral cerebral ventricle with fluorescein-labeled Abeta40 (FAb). Hypercholesterolemic rabbits had reduced brain efflux of FAb, supporting a possible physiologic mechanims linking AVD and AD.

Type of Paper: Article
Title: Terminal Continuation (TC) RNA Amplification Enables Expression Profiling Using Minute RNA Input Obtained From Mouse Brain
Authors: Melissa J. Alldred 1, Shaoli Che 1, 2 and Stephen D. Ginsberg 1, 2, 3,*
Affiliations: 1 Center for Dementia Research, Nathan Kline Institute, Orangeburg, NY; 2 Department of Psychiatry; 3 Physiology & Neuroscience, New York University School of Medicine, Orangeburg, NY
Abstact: A novel methodology named terminal continuation (TC) RNA amplification has been developed to amplify RNA from minute amounts of starting material. Utility of the TC RNA amplification method is demonstrated with two new modifications including obviating the need for second strand synthesis, and purifying the amplification template using column filtration prior to in vitro transcription (IVT). Using four low concentrations of RNA extracted from mouse brain (1 ng, 10 ng, 25 ng, and 50 ng), one round TC RNA amplification was compared to one round amplified antisense RNA (aRNA) in conjunction with column filtration and drop dialysis purification. The TC RNA amplification without second strand synthesis performed extremely well on custom-designed cDNA array platforms, and column filtration was found to provide higher positive detection of individual clones when hybridization signal intensity was subtracted from corresponding negative control hybridization signal levels. Results indicate that TC RNA amplification without second strand synthesis, in conjunction with column filtration, is an excellent method for RNA amplification from extremely small amounts of input RNA from postmortem human brain and mouse brain, and is compatible with microaspiration strategies and subsequent microarray analysis.

Published Papers:
 
(none)
 

International Journal of Molecular Sciences (ISSN 1422-0067)
Last change: 17 September 2008
© 2008 by MDPI, Basel, Switzerland